D

D tier · weak

The famous 63% figure is a maximum, not a mean, from an unpublished supplier in-house study in 17 women over 28 days, and no peer-reviewed trial has ever tested acetyl octapeptide-3 as a single active - the only two published human studies containing it are multi-ingredient microneedle patches. ClinicalTrials.gov returns zero registered studies, and the "topical botox" framing rests on a SNARE-competition mechanism never demonstrated in human skin.

snap-8

COSMETIC INGREDIENT · SNAP-25 FRAGMENT · TOPICAL NEUROMUSCULAR CLAIM

also: SNAP-8 · acetyl octapeptide-3 · acetyl glutamyl heptapeptide-1 · acetyl glutamyl octapeptide-3 · Ac-EEMQRRAD-NH2

SNAP-8 is an octapeptide corresponding to a fragment of SNAP-25, the SNARE protein required for acetylcholine vesicle fusion, acetylated at the N-terminus and amidated at the C-terminus. It carries the INCI name Acetyl Octapeptide-3 (also encountered as acetyl glutamyl heptapeptide-1) and was introduced by Lipotec as a topical alternative to botulinum toxin, extending the shorter and better-studied Argireline sequence by two residues. Its efficacy record consists of supplier in-house profilometry rather than peer-reviewed trials.

// we supply this one

available as a research reagent

≥ 98% HPLC · lyophilised powder · batch certificate published. Grade D above is our own and is not adjusted because we stock it.

from £19.95

per 10 mg

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the explanation

SNAP-8 is a lab-made copy of a small part of a protein that nerves use to release the signal telling muscles to contract. It is sold in creams as a needle-free alternative to Botox. The famous claim that it cuts wrinkle depth by 63% comes from the company that sells it, was the best single result rather than the average, and has never been published in a scientific journal or repeated by anyone independent.

regulatory status

Regulated cosmetic ingredient in the UK and EU; not a licensed medicine anywhere

Acetyl Octapeptide-3 is a listed INCI cosmetic ingredient with declared functions of humectant and skin conditioning, lawfully used in leave-on skincare in Great Britain under the retained UK cosmetics regime and in the EU and Northern Ireland under Regulation (EC) No 1223/2009, subject to a Cosmetic Product Safety Report and Product Information File. That clearance is for topical application to intact skin; the cosmetics framework excludes products intended for ingestion, inhalation, injection or implantation, and MHRA Guidance Note 8 weighs product form and claims together in deciding whether something is a medicinal product - a peptide marketed as a substitute for a prescription neurotoxin is close to that boundary. There is no MHRA, EMA or FDA approval of the peptide as a medicine. It is not named on the current WADA Prohibited List, but the S0 non-approved-substances clause would capture any substance without approval for human therapeutic use if presented for systemic use, and it has no documented status as a lawful dietary-supplement ingredient in the UK or US.

how it works · proposed mechanism

Botulinum toxin type A works intracellularly, cleaving SNAP-25 inside the presynaptic terminal of the motor neuron so that acetylcholine-containing vesicles cannot dock and fuse. SNAP-8 is a fragment of SNAP-25 itself, and the supplier's proposal is that it competes with the native protein for its place in the SNARE complex, blunting exocytosis. The whole claim therefore depends on a hydrophilic, multiply charged octapeptide reaching the inside of a nerve terminal beneath the dermis.

SNARE competition, as claimed by the supplier

Lipotec's product literature states that the peptide competes with the native protein for a position in the SNARE complex that is essential for muscle contraction, reducing neuronal excitability and so decreasing expression wrinkles. That is a mechanistic assertion in a commercial product sheet, not a published experiment. No electrophysiology, no vesicle-fusion assay and no immunohistochemistry in human skin is offered anywhere in the peer-reviewed literature for this specific peptide.

Relationship to Argireline: an extension, not an improvement

Acetyl hexapeptide-8 (Argireline, Ac-EEMQRR-NH2) covers the same SNAP-25 region and has multiple published anti-wrinkle studies; SNAP-8 simply adds Ala-Asp to the C-terminus. The reputation of the class is Argireline's, and SNAP-8's positioning as the better version rests on a supplier head-to-head comparison rather than on independent data. Notably, in the one independent randomised trial that tested acetylhexapeptide-3 topically, instrumental endpoints were non-significant.

The delivery problem the mechanism ignores

The peptide carries three acidic and two basic side chains and no lipid anchor, giving it a large polar surface and negligible partition into the stratum corneum. Acetylation and amidation improve resistance to amino- and carboxypeptidases but do nothing for permeation. Getting from the skin surface into the cytosol of a facial motor neuron requires crossing the stratum corneum, the epidermis, the dermis and a neuronal membrane - a sequence that has never been demonstrated for this molecule.

Why the microneedle studies do not rescue it

The only two peer-reviewed human studies containing acetyl octapeptide-3 delivered it through dissolving hyaluronic acid microneedle patches alongside other actives: ascorbyl glucoside and cyclic lysophosphatidic acid in a 24-subject study, and arginine/lysine polypeptide, palmitoyl tripeptide-5, adenosine and seaweed extracts in the other. Both bypass the stratum corneum mechanically and neither isolates the peptide's contribution. Their results cannot be attributed to SNAP-8 and say nothing about a cream.

together → The mechanism is a real molecular story about a real protein - SNAP-25 and the SNARE complex are well characterised, and botulinum toxin proves the target matters. What the mechanism does not establish is that a charged octapeptide applied to the skin surface reaches that target, that it competes effectively if it does, or that the resulting change is visible. It establishes nothing whatever about an injected or systemic route, for which no data of any kind exist.

what’s reported

0peer-reviewed clinical trials of acetyl octapeptide-3 as a single active
0registered studies on ClinicalTrials.gov
2peer-reviewed human studies containing it, both multi-active microneedle patches
17women in the unpublished supplier study behind the headline efficacy claim
63%supplier figure for wrinkle-depth reduction, reported as a maximum value rather than a mean
2PubMed records in total for the exact term acetyl octapeptide-3

evidence shape

7 sources

The composition of the cited literature by study type. This describes the shape of the evidence, not its quality — and it does not by itself set the grade.

official / regulatory4
randomised trials3
observational0
reviews0
preclinical0

⚠ the catch

The number that sells this peptide is not a trial result. Lipotec's product sheet describes an in-house study in 17 women over 28 days, with silicone imprints around the eyes analysed by confocal profilometry, and reports that the maximum reduction value of wrinkle depth was 63% - a best-case single measurement, not a mean, not placebo-controlled in any described way, and never published or peer-reviewed. A PubMed search for the exact term acetyl octapeptide-3 returns two records, both dissolving-microneedle patch studies in which the peptide is one of several actives delivered through a mechanically breached stratum corneum, so neither can attribute anything to SNAP-8. ClinicalTrials.gov returns zero registered studies. The peptide's reputation is largely inherited from the shorter Argireline sequence, which does have published trials - and in the one independent randomised topical trial of acetylhexapeptide-3, corneometry, TEWL and cutometry were all non-significant. Selling this compound as a lyophilised vial compounds the problem: even the weak evidence that exists is topical-only, and a charged octapeptide with no lipid anchor has no demonstrated route from anywhere to a motor nerve terminal.

key published findings

  • Lipotec product literature (COSSMA product information): 17 women, 28 days, silicone imprints around the eyes analysed by confocal profilometry, maximum wrinkle-depth reduction reported as 63%; no peer-reviewed publication is cited anywhere in the document.
  • PubMed contains exactly two records for the exact phrase acetyl octapeptide-3, and neither tests it alone.
  • Shin 2024 (Ann Dermatol): dissolving microneedle patch containing acetyl octapeptide-3 with ascorbic acid 2-glucoside and sodium cyclic lysophosphatidic acid in 24 subjects over 28 days improved eye wrinkles and elasticity versus placebo - a multi-active patch, not a SNAP-8 cream.
  • Avcil 2020 (J Cosmet Dermatol): hyaluronic acid microneedle patch loaded with arginine/lysine polypeptide, acetyl octapeptide-3, palmitoyl tripeptide-5, adenosine and seaweed extracts reduced fine lines by 25.8% and increased dermal density by 14.2%; the contribution of any single peptide is unrecoverable.
  • Aruan 2023 (J Clin Aesthet Dermatol): in the closest independent randomised test of the shorter parent analogue, acetylhexapeptide-3 cream produced non-significant corneometer, tewameter and cutometer results (p>0.05) over 8 weeks in seven subjects per arm.
  • ClinicalTrials.gov returns zero registered studies for SNAP-8, acetyl octapeptide or acetyl glutamyl heptapeptide.

limitations of the evidence

  • The headline efficacy figure is a maximum rather than a mean, from a supplier document with no protocol, randomisation, blinding or statistics disclosed, and no peer review.
  • No published study has tested acetyl octapeptide-3 as the only active variable, so no effect can be attributed to it.
  • Both peer-reviewed studies containing it used microneedle patches that bypass the stratum corneum, which is precisely the barrier a cream must cross.
  • The claimed SNARE-competition mechanism has never been demonstrated in human skin, and no human electrophysiological, neurophysiological or histological endpoint has been reported for the peptide.
  • Argireline, the shorter analogue whose reputation SNAP-8 trades on, is a different molecule with its own trial record, and even that record is mixed - the one independent randomised topical test of acetylhexapeptide-3 was null on instrumental measures.
  • Physicochemistry argues against the marketing: a multiply charged octapeptide with no lipidation has poor prospects of transdermal delivery to a presynaptic compartment, and no permeation study for this peptide has been published.

documented safety signals

  • Topical tolerability appears good in the limited record: no adverse effects were reported over 28 days in the 24-subject microneedle patch study, and the patch study in the other publication reported excellent tolerability.
  • As a listed INCI cosmetic ingredient it sits within the UK and EU cosmetics safety framework, which requires a qualified safety assessor's report for each finished topical product - a safety clearance for skin contact, not evidence of pharmacological safety.
  • No irritation, sensitisation or systemic toxicity dataset specific to acetyl octapeptide-3 is publicly available in a regulatory safety assessment comparable to the CIR palmitoyl peptide reviews.
  • There are no safety data of any kind for injection, ingestion or inhalation; absence of reported harm by those routes reflects absence of study, not evidence of safety, and the product is marketed with a neuromuscular mechanism claim that would be a medicinal claim if it were true.

identity

full nameAcetyl octapeptide-3 (Ac-Glu-Glu-Met-Gln-Arg-Arg-Ala-Asp-NH2), an N-acetylated, C-amidated octapeptide fragment of SNAP-25
categoryCosmetic
modalitypeptide
formulaC41H70N16O16S
molar mass1075.2 g/mol
cas868844-74-0
sequenceAc-Glu-Glu-Met-Gln-Arg-Arg-Ala-Asp-NH2

laboratory handling

storageLyophilised powder is stored desiccated at -20 °C and protected from light. Once reconstituted, laboratory practice is refrigeration at 2-8 °C for short-term use, or aliquoting and freezing to avoid repeated freeze-thaw cycles. Handling information only; this material has no approved human use.
solubilityShort hydrophilic peptide, readily soluble in sterile or bacteriostatic water and in dilute aqueous buffer. Lot-specific solubility is stated on the certificate of analysis.
co-studied withMarketed almost exclusively in multi-peptide formulations, frequently with acetyl hexapeptide-8, palmitoyl peptides or hyaluronic acid, and in microneedle patches; because no study isolates it, none of those combination results can be read as evidence for this peptide.

Handling information describes laboratory practice for a research reagent. It is not a preparation guide for use in humans.

the receipts

7 cited
MHRA Guidance Note 8: A guide to what is a medicinal product

Medicines and Healthcare products Regulatory Agency · 2025 · official

Acetyl Octapeptide-3 (CID 76283482): sequence, formula, mass and registry identifiers

PubChem, National Library of Medicine · 2026 · reference

snap-8 product information (mechanism and in-house in vivo profilometry data)

Lipotec, published in COSSMA · 2010 · press release

Sources marked tertiary or press release are the weakest citations on this page.

others in Cosmetic

Research use only. snap-8 is catalogued here as a research reagent. Nothing on this page is medical advice, a treatment recommendation, or a dosing protocol. This compound is not for human consumption.